Uncertain gait causes. Gait disturbance (dysbasia)

Almost all older people suffer from various disorders in the functioning of the body. These symptoms are so common that many of us consider them completely natural. But in fact, any disturbances in well-being, appearance and behavior may be the first signs of serious health problems. Therefore, under no circumstances should they be ignored. The usual may indicate pathological processes that pose a threat to life and health. Let's discuss problems with gait, tell you what a shuffling gait may indicate, consider the causes and its treatment in the elderly.

Causes of shuffling gait in old age

Shuffling in old age can be explained by previous injuries to the spine, hip joint, kneecap etc. Such a gait disturbance sometimes indicates an impending stroke, a pinched nerve, or a neuromuscular disorder.

Developing Parkinson's disease is also considered a possible explanation for shuffling in old age. This is a fairly serious neuromuscular disease. With this problem, patients begin to walk with small steps, their gait often becomes unsteady. The patient seems to be dragging his legs behind him.

The same shuffling gait in older people can also be observed with dementia, including with. In this case, foot dragging is explained by problems with thinking. However, with dementia, shuffling is preceded by other symptoms - disturbances in memory, thinking, etc.

Shuffling gait – treatment:

Treatment for shuffling gait in older adults depends on many factors. This disorder is not a separate disease, but only a symptom. Therefore, when it appears, you need to seek doctor’s help to identify the reasons that caused the shuffling.

It is considered a fairly serious and common cause of such gait disturbance. This is a chronic progressive disease of the central nervous system, in which there is damage to the neurons of the substantia nigra in the brain. Treatment for this pathological condition is aimed at eliminating the symptoms of the disease and prolonging the active life of patients.

Modern medications for Parkinson's disease need to be taken daily. The dosage is selected individually exclusively by the doctor.

Most often, patients with this diagnosis are prescribed levodopa; they effectively compensate for the lack of dopamine in the brain. However, prolonged use of such drugs often leads to the development of complications, for example, involuntary movements - dyskinesia. Patients have to come to terms with them.

Also, treatment of Parkinson's disease may involve taking drugs from other groups - dopamine agonists, amantadines, COMT inhibitors. These medications are less effective than levodopa, but they can delay its use and reduce complications associated with its use.

If patients have a predominance of the symptom of trembling of the limbs, they are usually prescribed anticholinergic drugs, represented by cyclodol and akinetholone. Patients whose age has exceeded sixty years are discharged.

To slow down the death of neurons, the drugs PC-Merz, Yumex and are used.

Physical therapy exercises are also recommended for patients with Parkinson's disease. Such exercises are aimed at training walking and balance, they are performed under the supervision of an instructor.

Folk remedies from Parkinson's disease

Patients with Parkinson's disease may also benefit from traditional medicine. Before using them, you should definitely consult a specialist.

St. John's wort infusion - use for Parkinson's. So you can prepare three tablespoons of St. John's wort and brew them with seven hundred milliliters of boiling water. Leave for one night. In the morning, strain the medicine, divide it into four equal parts. Take the infusion half an hour before meals.

Oregano infusion - use for Parkinson's. The reception gives an excellent effect. Brew a teaspoon of dry herb with a glass of boiling water, then leave for five to ten minutes. Strain and drink as tea. The duration of such therapy is three months.

Fainting and paralysis in Parkinson's disease? If a patient with Parkinson's disease experiences fainting or paralysis, you need to combine equal parts of lavender, snakehead, oregano and. Grind the mixture into powder. Brew four tablespoons of this mixture with a liter of boiling water. After three hours, strain and take a third of a glass three times a day. It is best to take this medicine on an empty stomach.

Rosehip root decoction. For Parkinson's disease, you can also use medicine based on. Brew a couple of tablespoons of crushed raw materials with a glass of boiling water and simmer over low heat for twenty minutes. Cool this decoction and then strain it. Take half a glass half an hour before meals.

Sage infusion - use for Parkinson's. It will also help cope with the symptoms of Parkinsonism. Brew a couple of teaspoons of chopped herbs with half a liter of boiling water. After half an hour, strain the finished infusion. Take half a glass of it on an empty stomach. Repeat the intake four times a day.

A shuffling gait can be a completely natural symptom of aging in older people, but in some cases it indicates serious health problems that require targeted treatment under the supervision of a doctor.

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Walking- one of the most complex and at the same time common types of physical activity.

Cyclic walking movements trigger the lumbosacral centers of the spinal cord and regulate the cortex cerebral hemispheres, basal ganglia, brainstem structures and cerebellum. This regulation involves proprioceptive, vestibular and visual feedback afferentation.

Gait The human brain is a harmonious interaction of muscles, bones, eyes and inner ear. The coordination of movements is carried out by the brain and the central nervous system.

If there are disturbances in certain parts of the central nervous system, various movement disorders may occur: a shuffling gait, sudden jerking movements, or difficulties in bending joints.

Abasia(Greek ἀ- prefix with the meaning of absence, non-, without- + βάσις - walking, gait) – also dysbasia– disturbance of gait (walking) or inability to walk due to gross gait disturbances.

1. In a broad sense, the term abasia means gait disturbances with lesions involving different levels systems for organizing the motor act, and includes such types of gait disorders as ataxic gait, hemiparetic, paraspastic, spastic-atactic, hypokinetic gait (with parkinsonism, progressive supranuclear palsy and other diseases), apraxia of walking (frontal dysbasia), idiopathic senile dysbasia , peroneal gait, duck gait, walking with pronounced lordosis in the lumbar region, hyperkinetic gait, gait in diseases of the musculoskeletal system, dysbasia in mental retardation, dementia, psychogenic disorders, iatrogenic and drug dysbasia, gait disorders in epilepsy and paroxysmal dyskinesia.

2. In neurology the term is often used astasia-abasia, with integrative sensorimotor disorders, more often in the elderly, associated with a violation of postural or locomotor synergies or postural reflexes, and often a variant of imbalance (astasia) is combined with a walking disorder (abasia). In particular, frontal dysbasia (gait apraxia) is distinguished with damage to the frontal lobes of the brain (as a result of stroke, discirculatory encephalopathy, normal pressure hydrocephalus), dysbasia in neurodegenerative diseases, senile dysbasia, as well as gait disturbances observed during hysteria (psychogenic dysbasia).

What diseases cause gait disturbance?

A certain role in the occurrence of gait disorders belongs to the eye and inner ear.

Older people with deteriorating vision develop gait disturbances.

A person with an inner ear infection may exhibit balance problems that lead to disturbances in their gait.

One of the common sources of gait disturbances is functional disorders of the central nervous system. These may include conditions associated with sedatives, alcohol, and drug abuse. Poor nutrition appears to play a role in the development of gait disturbances, especially in older people. Vitamin B12 deficiency often causes numbness in the limbs and poor balance, leading to changes in gait. Finally, any disease or condition that affects nerves or muscles can cause gait disturbances.

One such condition is a pinched disc in the lower back. This condition is treatable.

More serious disorders that cause gait changes include amyotrophic lateral sclerosis (Lou Gehrig's disease), multiple sclerosis, muscular dystrophy, and Parkinson's disease.

Diabetes often causes loss of sensation in both legs. Many people with diabetes lose the ability to determine the position of their legs in relation to the floor. Therefore, they experience postural instability and gait disturbances.

Some diseases are accompanied by gait disturbances. If there are no neurological symptoms, the cause of the gait disorder is difficult to find out even for an experienced doctor.

Hemiplegic gait is observed with spastic hemiparesis. In severe cases, an altered position of the limbs is characteristic: the shoulder is adducted and turned inward, the elbow, wrist and fingers are bent, the leg is extended at the hip, knee and ankle joints. The step with the affected leg begins with abduction of the hip and movement in a circle, while the body deviates in the opposite direction (“the hand asks, the leg squints”).
With moderate spasticity, the position of the arm is normal, but its movements in time with walking are limited. The affected leg bends poorly and is turned outward.
Hemiplegic gait is a common residual disorder after stroke.

With a paraparetic gait, the patient moves both legs slowly and tensely, in a circle - the same as with hemiparesis. Many patients have legs that cross like scissors when walking.
Paraparetic gait is observed with spinal cord lesions and cerebral palsy.

Cock gait is caused by insufficient dorsiflexion of the foot. When stepping forward, the foot partially or completely hangs down, so the patient is forced to raise his leg higher - so that the toes do not touch the floor.
Unilateral disorder occurs with lumbosacral radiculopathy, neuropathy of the sciatic nerve or peroneal nerve; bilateral - for polyneuropathy and lumbosacral radiculopathy.

The duck gait is explained by the weakness of the proximal muscles of the legs and is usually observed with myopathies, less often with lesions of the neuromuscular junction or spinal amyotrophy.
Due to weakness of the hip flexors, the leg is lifted off the floor due to the tilt of the torso, the rotation of the pelvis promotes the movement of the leg forward. Weakness of the proximal leg muscles is usually bilateral, so the patient walks in a waddling manner.

With a parkinsonian (akinetic-rigid) gait, the patient is hunched over, his legs are bent, his arms are bent at the elbows and pressed to the body, and a pronation-supination rest tremor (with a frequency of 4-6 Hz) is often noticeable. Walking begins by leaning forward. Then follow mincing, shuffling steps - their speed steadily increases, as the body “overtakes” the legs. This is observed when moving both forward (propulsion) and backward (retropulsion). Having lost balance, the patient may fall (see "Extrapyramidal disorders").

Apraxic gait is observed in bilateral frontal lobe lesions due to impairment of the ability to plan and execute a sequence of actions.

Apraxic gait resembles Parkinsonian gait - the same “supplicant pose” and mincing steps - however, upon detailed examination, significant differences are revealed. The patient easily performs the individual movements necessary for walking, both lying and standing. But when he is asked to go, he cannot budge for a long time. Having finally taken a few steps, the patient stops. After a few seconds, the attempt to walk is repeated.
Apraxic gait is often associated with dementia.

With choreoathetotic gait, the rhythm of walking is disrupted by sudden, violent movements. Due to chaotic movements in the hip joint, the gait looks “loose.”

With a cerebellar gait, the patient places his legs wide apart, the speed and length of steps change all the time.
When the medial zone of the cerebellum is damaged, a “drunk” gait and ataxia of the legs are observed. The patient maintains balance both with open and closed eyes, but loses it when the position changes. The gait may be fast, but it is not rhythmic. Often, when walking, the patient experiences uncertainty, but it goes away if he is at least slightly supported.
When the cerebellar hemispheres are damaged, gait disturbances are combined with locomotor ataxia and nystagmus.

The gait with sensory ataxia resembles a cerebellar gait - legs widely spaced, loss of balance when changing position.
The difference is that when the eyes are closed, the patient immediately loses balance and, if not supported, may fall (instability in the Romberg position).

Gait of vestibular ataxia. With vestibular ataxia, the patient always falls to one side - regardless of whether he is standing or walking. There is obvious asymmetrical nystagmus. Muscle strength and proprioceptive sensation are normal - in contrast to unilateral sensory ataxia and hemiparesis.

Gait during hysteria. Astasia - abasia is a typical gait disorder during hysteria. The patient has preserved coordinated movements of the legs, both lying and sitting, but he cannot stand or move without assistance. If the patient is distracted, he maintains his balance and takes a few normal steps, but then defiantly falls - into the hands of the doctor or onto the bed.

Which doctors should I contact if gait disturbance occurs?

Neurologist
Traumatologist
Orthopedist
ENT

Hello. I am 24 years old, male. At the moment I am worried about: dizziness (unsteadiness when walking or when standing), objects around me do not rotate, only I sway like in a boat, some slight fog in the head, increased nervous tension, tension in the legs (not always). This all happens when it starts to wobble, it doesn’t wobble all the time, mostly on the street or outside the house, sometimes when I wake up, but it goes away after 20-30 minutes. This condition has lasted for 1.5 months. Since childhood I have had scoliosis and VSD (sweating, cold extremities). After a nervous breakdown 6 years ago (a relative died), tachycardia appeared, at the beginning it was strong, then after a year it decreased and I got used to it, as well as pain in the heart area .At that time I checked the heart: ECG, echocardiography, daily ecg, ecg with stress and everything was normal, only a slight tachycardia + donated blood for thyroid hormones (normal). The doctor diagnosed VSD. I gradually got used to it, then I went to the army.After physical activity and going uphill there were feelings of palpitations and shortness of breath, but they went away after a couple of minutes. I thought it was from VSD and didn’t attach any importance to it and everything was fine. This year, in March, I was poisoned or was it intestinal flu (the doctor didn’t really figure it out), he prescribed me antibiotics and bifidobacteria and everything seemed to go away, but the problems with the gastrointestinal tract remained (unstable stool, flatulence, rumbling) and problems with the gastrointestinal tract until now. I went to a gastroenterologist, but they didn’t find anything and diagnosed dysbacteriosis and urticaria. In May, I caught a cold in my neck, my neck hurt and my muscles pulled, I had daily pressing, squeezing headaches. I took an X-ray of my neck, diagnosed cervical osteochondrosis, and prescribed treatment. Gradually, headaches began to appear less frequently, now they practically do not bother me, they do happen, but rarely. At the end of September, my back and thoracic spine hurt, I took an x-ray and found S-shaped scoliosis, the initial manifestations of osteochondrosis. After a couple of weeks, my back began to go away and now it doesn’t hurt all the time, and sometimes it doesn’t hurt at all, but then these dizzinesses appeared - vacillation. Examinations that I have undergone from March 2012 to the present day: X-ray of the neck - initial signs of osteochondrosis (May 2012) Gastroscopy + Helicobacter test - gastroduodenitis and Helicobacter ++ - treated with antibiotics (May 2012) Kaprogram - normal General and complete biochemical blood test - normal , bilirubin is slightly increased (possibly from medications) (June 2012) General analysis was done 3 times this year, tests for HIV, hepatitis, sexually transmitted infections and hidden sexually transmitted infections - negative (June 2012) Ultrasound of the abdominal organs - no pathologies (June 2012 ) ECG is normal, slight tachycardia (June, September 2012) X-ray of the thoracic region - S-shaped scoliosis, initial signs of osteochondrosis (the neurologist also said kyphosis) (September 2012) Fluorography - without pathologies (September 2012) Ultrasound dopplerography of the vessels of the head and neck - in normal, but they found a slight pinching of the artery (they said it was due to osteochondrosis) (October 2012) MRI of the brain was normal, only there were arachnoid changes of a liquor cystic nature (a neurologist looked at the pictures and said it’s not scary, it doesn’t affect anything) (October 2012) In December 2011, I also did an EEG - normal pressure from 110-80 to 130-80. During all this time, the following diagnoses were made: Initial osteochondrosis, scoliosis, gastroduodenitis, ADHD, IBS and VSD. Now I’m worried about dizziness and unsteadiness, problems with the gastrointestinal tract, pain in the neck and back (not severe), increased nervous tension (besides, I’ve been going to the doctors all year, new symptoms keep popping up) The main thing is to find the cause of the unsteadiness, it’s really bothering me. With the gastrointestinal tract, I will resolve the issue with a gastroenterologist. The neurologist does not find any pathologies for me, he diagnoses osteochondrosis and all sorts of things. Is it really all from all kinds of diseases? What else can cause such a set of symptoms? What other examinations need to be done?

Unsteadiness of gait is not a diagnosis, but a symptom that serves as a manifestation of a disease. Therefore, when it appears, you should immediately consult a doctor for examination.

The main reason is vascular spasm, which is caused by tension in the muscles of the back, in particular the neck. This often happens with increased anxiety and tension. In this case, the main treatment should not be medication, but should be carried out through sessions with a psychotherapist.

Another common cause of unsteadiness is cervical osteochondrosis, which is diagnosed very often these days. Dizziness and darkening of the eyes may be added to this symptom. This happens due to the incorrect position of the vertebrae, which compress the vessels, and blood cannot flow into the brain in the required amount, which is why its cells are left without oxygen, which is reflected in the manifestation of a variety of symptoms.

However, the causes of unsteadiness when walking can be more serious. For example, diseases characterized by this symptom can be considered:

  1. Early cerebellar ataxia.
  2. Late neurosyphilis.
  3. Cerebellar tumors.
  4. Meningeal tuberculoma.
  5. Acute disseminated encephalitis.
  6. Candidal meningitis.
  7. Arnold-Cairo anomaly.
  8. Brain abscess.
  9. Drug-induced polyneuropathy.

But this is not the entire list of reasons that can cause unsteadiness in gait, so to make a correct diagnosis, as well as to prescribe treatment, you must definitely consult a doctor.

By type, this condition can be divided into systemic and non-systemic. The first type includes lesions of the vestibular and auditory apparatus, as well as diseases of the brain and spinal cord. Non-systemic manifestations are consequences of chronic diseases of the heart, blood vessels, endocrine and respiratory systems.

The essence of the disease

Unsteadiness of gait without dizziness is a common manifestation. This is a dystrophic disease of cartilage and bone tissue, which manifests itself mainly in adulthood. The basis of the disease is dystrophy of the intervertebral discs. All this negatively affects the blood supply, nutrition and hydration of the intervertebral disc. And the resulting deformation leads to a strong narrowing of the space between the vertebrae.

Due to the decrease in intervertebral space, spinal nerves may be pinched. The main symptoms of such infringement will depend on where it happened - in the cervical, chest or lumbar region spine. If it is the neck, then the main manifestations are headache, dizziness and other manifestations associated with malnutrition of the brain. If this is the thoracic part of the spine, pain in the heart area may appear, which resembles the symptoms of myocardial infarction or angina. And if it is the lower back, then the symptoms may be in the form of impaired mobility in the legs, their weakness and decreased skin sensitivity.

Therefore, dizziness and unsteadiness of gait can be the cause of such an insidious disease as osteochondrosis. Due to compression of the nerve root, the transmission of impulses through it to the muscles of the legs is disrupted, and this can be either one leg or both. This is the cause of weakness in the legs.

What to do

Unsteadiness of gait with osteochondrosis is a rather dangerous sign that you should immediately pay attention to and see a doctor. This is especially dangerous when weakness in the legs gradually increases. But before starting treatment, you need to undergo a comprehensive examination so that the doctor can understand what caused this pathology and why this symptom appeared.

If pain occurs, it should be relieved with painkillers. Here you can use not only one medicine, as well as a mixture of several components. For example, solutions could be:

  1. Analgin + no-spa + lasix + novocaine. All this is added to 150 ml of 0.9% sodium chloride solution and applied intravenously.
  2. Baralgin + Relanium + Dexazone + Novocaine. All this is introduced into a 5% glucose solution in an amount of 200 ml and also dripped as an intravenous infusion.
  3. Analgin + vitamin B12 + no-spa + reopirin. All drugs are mixed in one syringe and administered intramuscularly.

Unsteadiness of gait with osteochondrosis is a symptom that requires the prescription of other drugs, for example, pentoxifylline, which improves blood circulation, theonicol or nicotinic acid, and drugs that will stimulate the outflow of blood through the veins. Of course, we must also remember about the general strengthening effect of vitamin and mineral complexes, which help replenish the balance of nutrients in the body.

By the way, you may also be interested in the following FREE materials:

  • Free books: "TOP 7 harmful exercises for morning exercises that you should avoid" | “6 Rules for Effective and Safe Stretching”
  • Restoration of knee and hip joints with arthrosis- free video recording of the webinar conducted by a physical therapy and sports medicine doctor - Alexandra Bonina
  • Free lessons on treating low back pain from a certified physical therapy doctor. This doctor has developed a unique system for restoring all parts of the spine and has already helped more than 2000 clients with various back and neck problems!
  • Want to know how to treat a pinched sciatic nerve? Then carefully watch the video at this link.
  • 10 essential nutritional components for a healthy spine- in this report you will find out what it should be like daily diet so that you and your spine are always healthy in body and spirit. Very useful information!
  • Do you have osteochondrosis? Then we recommend studying effective methods treatment of lumbar, cervical and thoracic osteochondrosis without drugs.

I’m sure you always pay attention to a beautiful figure and a beautiful gait. Have you ever wondered what exactly ensures our beautiful gait?

Central nervous system: cerebral cortex, extrapyramidal and pyramidal systems, brain stem, spinal cord, peripheral nerves, cerebellum, eyes, vestibular apparatus of the inner ear and of course the structures that govern all this - skeleton, bones, joints, muscles. Healthy listed structures, correct posture, smoothness and symmetry of movements ensure normal gait.

Gait is formed from childhood. Congenital dislocations of the hip joint or joint can subsequently lead to shortening of the limb and gait disturbance. Hereditary, degenerative, infectious diseases of the nervous system, manifested by muscle pathology, impaired tone (hypertonicity, hypotonicity, dystonia), paresis, hyperkinesis, will also lead to gait disturbances - cerebral palsy, myopathies, myotonia, Friedreich's disease, Strumpel's disease, Huntington's chorea, polio .

Properly selected shoes will influence the formation of the correct gait. With tight shoes, the child will curl his toes, the formation of the arch of the foot will be disrupted, the joints may become deformed, resulting in arthrosis of the joints and gait disturbance. Flat feet and club feet interfere with gait. Incorrect long-term sitting at a desk will lead to curvature of the spine (scoliosis) and gait disturbance.

When walking correctly, the torso should lean slightly back. You need to keep your back straight, your chest straight, and your buttocks taut. With each step, your feet should be placed in one line, with your toes turned outward. Keep your head slightly elevated. Look straight ahead or slightly up.

Damage to the peripheral nerves - peroneal and tibial - will lead to gait disturbance. “Stepping” - when walking, the foot “slaps” because dorsal flexion (bending) is impossible and the foot hangs down. When walking, a patient with damage to the peroneal nerve tries to raise his leg higher (so as not to touch the floor with his toes), the foot hangs down, and when lowering his leg resting on the heel, the foot slaps on the floor. This type of gait is also called “rooster gait.” The peroneal nerve is affected by compression-ischemic, traumatic, toxic neuropathies. Compression means that you have compressed a nerve and/or blood vessels and developed ischemia - circulatory failure. This is possible, for example, when sitting for a long time: “squatting” - repairs, gardening; in small buses on long trips. Sports activities, very sound sleep in an awkward position, tight bandages, plaster splints can cause circulatory problems in the nerves.

Damage to the tibial nerve makes it impossible to plantar flex the foot and toes and turn the foot inwards. In this case, the patient cannot stand on his heel, the arch of the foot deepens, and a “horse” foot is formed.

Atactic gait– the patient walks with his legs spread wide apart, leaning to the sides (usually towards the affected hemisphere), as if balancing on an unstable deck, the movements of the arms and legs are not coordinated. Turning the body is difficult. This is a "drunk walk". The appearance of an ataxic gait may indicate a disorder vestibular apparatus, about circulatory disorders in the vertebrobasilar basin of the brain, about problems in the cerebellum. Vascular diseases, intoxications, and brain tumors can manifest as an ataxic gait and even frequent falls.

Antalgic gait– with radicular pain syndromes of osteochondrosis, the patient walks, curving the spine (scoliosis appears), reducing the load on the diseased root and thereby the severity of pain. When there is pain in the joints, the patient spares them, adapting his gait to reduce the pain syndrome - lameness appears, and with coxarthrosis, a specific “duck” gait - the patient waddles from foot to foot like a duck.

With damage to the extrapyramidal systems, Parkinsonism develops akinetic-rigid syndrome– movements are constrained, muscle tone is increased, the coordination of movements is impaired, the patient walks, bent over, tilting his head forward, bending his arms at the elbow joints, in small steps, slowly “shuffling” along the floor. It is difficult for the patient to start moving, “disperse” and stop. When stopped, it continues for some time to move unsteadily forward or to the side.

With chorea it develops hyperkinetic-hypotonic syndrome with violent movements in the muscles of the trunk and limbs and periods of muscle weakness (hypotonia). The patient walks with a sort of “dancing” gait (Huntington’s chorea, St. Vitus’s dance).

When the pyramidal system is damaged in various diseases of the nervous system, paresis and paralysis of the limbs. Thus, after a stroke with hemiparesis, a characteristic Wernicke–Mann position is formed: the paralyzed arm is brought to the body, bent at the elbow and wrist joint, the fingers are bent, the paralyzed leg is maximally extended at the hip, knee, and ankle joints. When walking, the impression of an “elongated” leg is created. The patient, in order not to touch the floor with his toe, moves his foot in a semicircle - this gait is called “circumducing.” In milder cases, the patient limps, muscle tone in the affected limb is increased and therefore there is less flexion in the joints when walking.

In some diseases of the nervous system, it may develop lower paraparesis- weakness in both legs. For example, with multiple sclerosis, myelopathies, polyneuropathies (diabetic, alcoholic), Strumpel's disease. With these diseases, gait is also impaired.

Heavy gait– with swelling of the legs, varicose veins, poor circulation in the legs - a person stomps heavily, with difficulty lifting his burning legs.

Gait disturbances are always a symptom of some disease. Even a common cold and asthenia changes gait. A lack of vitamin B12 can cause numbness in the legs and affect gait.

Which doctor should I contact if I have gait problems?

If there is any disturbance in gait, you need to consult a doctor - a neurologist, traumatologist, therapist, otolaryngologist, ophthalmologist, angiosurgeon. It is necessary to be examined and treated for the underlying disease that caused the gait disturbance or to adjust the lifestyle, the habit of sitting cross-legged at a table, and to diversify a sedentary lifestyle with activities physical culture, visiting the pool, fitness classes, water aerobics, walks. Courses of multivitamins of group B and massage are useful.

Consultation with a doctor regarding gait disorders:

Question: how to sit at the computer correctly so as not to develop scoliosis of the spine?
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